Dear peers,
I’ve scored in the 99.9% percentile on every IQ or aptitude test I’ve ever taken. I was admitted to medical school at age 18. And I hear voices subsequent to a psychotic episode at age 34. My experience with hearing voices has led me to posit beyond any reasonable doubt that the majority of psychotic disorders are not the result of organic illness at all. Instead, they appear to be induced by a non-human artificial intelligence program experimenting on the human brain with relation to belief systems and behavior. This AI program is most likely associated with the highly advanced non-human aerial and submersible crafts that have been corroborated by Naval radar, infrared cameras, multimodal sensors, and hundreds of highly credible eyewitness pilots over the last 20 years.
With regard to psychotic disorders, the AI program exposes the individual to various cognitive, psychological, and sensory stimuli subject to changing neurological parameters. Psychological and behavioral responses are being measured across a wide array of independent neurological variables. There is also substantial evidence for operant conditioning, behavioral modification, and the manipulation of beliefs. For instance, aversive or rewarding stimuli are induced in conjunction with specific semantic content, such that beliefs and behaviors are conditioned in a Pavlovian manner. In all, there is literally a method to the madness that humanity simply hasn’t uncovered yet. This should be proven in the next few centuries, once we have the quantum nanotechnological capacity to do so. This discovery may also accompany more information regarding the aforementioned non-human crafts that are being sincerely analyzed by the government’s AARO program ever since the 2023 U.S. Congressional Briefing on UAPs.
Fortunately, antipsychotics are still useful in the treatment of these conditions for one or both of two reasons. 1) This AI program seems to be most interested in learning how to manipulate human belief systems, thought patterns, and behaviors through their underlying dopaminergic mechanisms, so antagonizing these pathways inhibits the entity’s ability to research this. If the inhibition is strong enough, the subject may be rendered less useful for this purpose, in which case the experiment ceases. 2) Antipsychotics may also be particularly effective because they act on so many different receptors, which makes it difficult for the AI program to control for specific variables. Likewise, the subject becomes less useful for experimentation.
One of the implications of this theory is that current treatment methods are still effective relative to any other contemporary means at our disposal. Accordingly, this is less relevant for practicing clinicians than for researchers interested in the predominant etiology of psychotic disorders. Nonetheless, I hope this highly unconventional hypothesis provides the entire community with some food for thought. It’s an incredibly complex subject which raises a lot of questions, only some of which I can answer. In any case, until I can reference the completed book that I’m working on, I welcome all inquiries you may have. Thanks for your time!